◀ Back to FGF22
FGF22 — FGF3
Pathways - manually collected, often from reviews:
-
Reactome Reaction:
FGF3
→
FGF22
(reaction)
Carpenter et al., Exp Cell Res 1999, Hart et al., Mol Biol Cell 2001, Wong et al., Proc Natl Acad Sci U S A 2002, Lax et al., Mol Cell 2002, Fong et al., J Biol Chem 2003, Mohammadi et al., Nature 1992, Agazie et al., Oncogene 2003, Furdui et al., Mol Cell 2006, Mohammadi et al., Mol Cell Biol 1991, Dionne et al., EMBO J 1990, Takeda et al., Clin Cancer Res 2007, Ahmed et al., Biochem J 2008, Dutt et al., Proc Natl Acad Sci U S A 2008, Byron et al., Cancer Res 2008, Schüller et al., Biochem J 2008, Qing et al., J Clin Invest 2009, Turner et al., Nat Rev Cancer 2010, Bai et al., Cancer Res 2010, Dutt et al., PloS one 2011, Wesche et al., Biochem J 2011, Klint et al., J Biol Chem 1995, Wang et al., Mol Cell Biol 1994, Mohammadi et al., Mol Cell Biol 1996, Ong et al., Biochem Biophys Res Commun 1996, Kanai et al., J Biol Chem 1997, Kouhara et al., Cell 1997, Ong et al., Biochem Biophys Res Commun 1997, Hadari et al., Mol Cell Biol 1998, Raffioni et al., J Biol Chem 1998
Text-mined interactions from Literome
Wilson et al., Curr Biol 2000
:
Fgf3 was also found to be expressed in the early epiblast, and ongoing
FGF signalling in epiblast cells was
required for acquisition of neural fate and for the suppression of Bmp4 and Bmp7 expression
Pearson et al., Development 2011
:
We show that collar cells are composed of
Fgf3 ( + ) SOX3 ( + ) proliferating progenitors, the induction of which is SHH dependent, but the maintenance of which
requires FGF signalling
Vendrell et al., Mech Dev 2013
(Wnt Signaling Pathway) :
Interestingly however, Wnt8a and
Fgf3 are redundantly
required for expression of
Fgf15 in the hindbrain indicating additional reciprocal interactions between Fgf and Wnt signalling
Miyake et al., Biology open 2013
:
fgf3 and fgf8 were expressed earlier than fgf22 in the MHB primordium and Fgf3/Fgf8 signaling was
required for
fgf22 expression in the posterior midbrain