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BCL10 — NFKBIA

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: BCL10 — NFKBIA (physical association, affinity chromatography technology) Carvalho et al., J Cell Sci 2010*

Text-mined interactions from Literome

Tsukahara et al., J Virol 1999 : Furthermore, Tax induced transactivation of the bcl-x promoter was also diminished by the mutant IkappaBalpha , which specifically inhibits NF-kappaB activity
Bui et al., J Cell Biol 2001 : Activation of nuclear factor kappaB and Bcl-x survival gene expression by nerve growth factor requires tyrosine phosphorylation of IkappaBalpha
Tran et al., J Biol Chem 2005 (Astrocytoma...) : Furthermore Fn14 induced transactivation of the BCL-X promoter was also diminished by the super-repressor IkappaBalpha mutant, which specifically inhibits NFkappaB activity, and by mutations in the NFkappaB binding motif of the BCL-X promoter
Xu et al., Oncogene 2005 (Prostatic Neoplasms) : Analogous to treatments with SFN and PEITC, SR-IkappaBalpha also strongly inhibited NF-kappaB transcriptional activity as well as VEGF, cylcin D1, and Bcl-X ( L ) expression
Klemm et al., Proc Natl Acad Sci U S A 2007 : By using murine embryonic fibroblasts from Bcl10- or Malt1-deficient mice as a genetic model, we report that Bcl10 and Malt1 are critically required for the degradation of IkappaB-alpha and the subsequent NF-kappaB induction in response to LPA stimulation
Bhattacharyya et al., Inflamm Bowel Dis 2009 : DSS induced increases in phospho-IkappaBalpha , nuclear NFkappaB ( p65 ), and IL-8 secretion in human colonic epithelial cells in tissue culture are attributable to a reactive oxygen species ( ROS ) -induced pathway of inflammation, and do not require TLR4, MyD88, or Bcl10 , which are associated with the innate immune pathway of NFkappaB-IL-8 activation
Borthakur et al., Inflamm Bowel Dis 2010 : Bcl10 was required for PAF induced I kappaB alpha phosphorylation, NF-kappaB activation, and IL-8 production in NCM460, a cell line derived from normal human colon, and Caco-2, a transformed human intestinal cell line